Just some fun, peer reviewed reads:

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7395818/

 https://pubmed.ncbi.nlm.nih.gov/21079686/

https://pubmed.ncbi.nlm.nih.gov/21079686/

tl:dr:  Zinc and some commonly available OTC supplements that act as ionophores screw with coof, and other viri being able to replicate.  I think a lot of us took zinc when we had a cold or whatnot, and it sorta helped, maybe didn't, but there seems to be a missing ingredient.  Not missing now. 

 

I'm just going to spell some things out, no funny.  If you read my earlier post about coof, you know I'm of the opinion that if you had it, you're done.  Let me expand on why.

All the variants of concern involve drift / mutations on the S-1 protein - that's how it grabs onto ACE-2 receptors, uses the furin unzipper to unzip the cell wall, and the S-2 protein to fuse the nucleocapcid into a cell.  tl:dr:  there is a protein on Coof that anchors on a receptive part of some cells, another part that is a can opener, and a third part that facilitates the virus taking a big crap of it's payload inside cellular membranes.  That's kinda how viruses work, but anywho. 

If you already had it and got over it, your body has seen S-1 the grabber.  Also S-2, and another dozen or so proteins on the viral shell, and it will have an immune response too all of them unless you are immunocompromised.

Drift in in the S-1 protein isn't meaningful for people who had it already.  It may be for people who have an entire immune response that relies on S-1 protein antibodies produced through a vaccine, but I can't fix that.  I can't fix the potential for ADE, etc.

 If you already had Covid and naturally recovered, the shots don't provide any benefit

https://sharylattkisson.com/2021/08/covid-19-natural-immunity-compared-to-vaccine-induced-immunity-the-definitive-summary/

This is probably the most interesting article I've read today:

Natural Immunity

 CD4+T cells seem to be the moderator between sniffy Co-V2 and the bad stuff.  The CD4+T cells in some people have cross reactivity, meaning at least partial recognition, of coof from prior Corona type virus exposure, and can lead a moderated response - cross reactivity.  CD8+T cells get called early, B4 cells also get the message, and coof gets squelched through a mild cytokine and robust humoral [antibody] response.  This is moderated by interferon timing that differs if there isn't an existing CD4+T cell response.

 Some people express ACE2 receptors on CD4+T cells, and Co-V2 can infect those cells.  If the CD4+T cells are infected by Co-V2, they don't message the CD8+T or B4 cells into action the same way.  Co-V2 has more time to set up shop because part of the natural immune response isn't working, and it infects way more cells.  The large number of dying cells creates a different interferon response that may turn into a cytokine storm.